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Bacterial CM and DCM utilization starts with dehalogenation, causing diverse physiological stresses such as production of intracellular HCl, which lowers pH and increases ionic strength, and formation of DNA adducts (Kayser and Vuilleumier, 2011

LMP1 inhibits MDM2-mediated p53 ubiquitination associated with p53 Ser20 phosphorylation and also induces K63-linked ubiquitination of p53 by interacting with TRAF2, thus disrupting tumor cell apoptosis and cell cycle arrest