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Unknown factors include potential hormonal effects, cellular signaling imbalance, and unintended tissue growth responses after prolonged exposure

Mitochondria, as the core source and main target of ROS [449], their functional damage will further form a positive feedback loop of ROS generation - mitochondrial damage, causing energy metabolism disorders and inducing mitochondria to release pro-apoptotic factors such as cytochrome C, ultimately leading to neuronal apoptosis [450]

We discovered an increase in mitochondrial mass, spare respiratory capacity (SRC)both considered hallmarks of T mem cellsand, indeed, an enrichment of T memory cell formation in vivo and in vitro (van der Windt et al , 2012, 2013)