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10.1016/j.jhepr.2022.100532 62

Patients with BD demonstrate both central and peripheral elevations in proinflammatory elements (cytokines, chemokines, prostaglandins, acute-phase reactants, oxidative/nitrosive species), increased inflammatory gene expression, as well as aberrant cellular (T-cell, monocyte, microglial) and complement activation [2, 4, 5]