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1k), suggesting that CAR T cells from LTR preferentially shunt HK glycolytic metabolites to the pentose phosphate pathway (PPP) rather than glycolysis, and by CPT1A could convert long-chain fatty acids into acylcarnitines for shuttling into the mitochondrial matrix for subsequent oxidation, previously associated with increased basal and maximal mitochondrial respiration 5,6,7,8

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Glutamine is important for TLR-induced activation of human plasmacytoid DCs but TLR-stimulation induces glycolysis in CD1c + mDC ( Glutamine metabolism differs between different types of immune cells and is determined by the activation and differentiation status of the respective cell type ( via glutaminolysis is important for alternative M2-like activation ( In our study glutamine deprivation itself did not alter cytokine secretion or surface antigen expression neither in human monocyte-derived DC nor in macrophages