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The intricate crosstalk among mitochondrial dysfunction, ferroptosis, inflammasomes, and endoplasmic reticulum (ER) stress creates a pathological network that underpins ovarian dysfunction, metabolic abnormalities, and chronic inflammation in PCOS, highlighting promising novel targets for diagnosis and therapeutic intervention in this complex disorder

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2B left, in green, Supplementary Table 2) reduced the biomass production to zero in most - but not all - IN-HOUSE models without significantly affecting the control models (Fig

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