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We do not anticipate any significant increase in utilization or payment impact for additional dental services given the historically low utilization of these therapies

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Taken together, these results indicate that unrestrained fork progression induced by fork reversal deficiency in HRASV12/cyclin E1-overexpressing cells is dependent on PRIMPOL and further support a role for PRIMPOL in restarting R-loop-stalled forks via the MUS81-LIG4-ELL axis