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Notably, we demonstrate that partial inhibition of v-ATPase confers metabolic resilience across diverse models of mitochondrial dysfunction, including primary models of mitochondrial translation defects

But by normalizing the cytokine environment and supporting proper T-cell differentiation, it may create conditions more favorable for tolerance induction

Concerning ABCB4 disease with at least one missense variant and a clinical phenotype with symptoms of hereditary cholestasis such as ICP, gallstone disease, LPAC ACC, DIC, UDCA therapy is recommended [91]

Experiment 3 Eighteen animals were assigned to 6 dietary groups ( n = 3)