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Conclusion: Following molecular diagnosis, clinicians can implement personalized treatment benefiting patients, as the literature already establishes that most of the individuals with GCK-MODY have good glycemic control with diet alone, without pharmacological treatment, and those with HNF1A mutations respond well to low-dose sulfonylureas

This underscores the need for future research to focus on detailed mechanism analysis, systematically evaluate its bidirectional regulatory effects, and avoid therapeutic risks stemming from a one-sided pursuit of single targets

PE might enable membrane destruction in other ways such as facilitating interactions between proteins and bilayer taking place due to PEs small polar head, or it might serve as a buffering agent to prevent excessive ferroptosis until overwhelmed by the presence of lipid peroxides 10

This substance initiates the active synthesis of collagen, which aids in restoring DNA chains and enhancing the production of the skin's own elastin