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aureus survives antibiotic treatment inside mammalian cells, C57BL/6J mice were infected via tail vein (i.v.) with the reporter strains and treated with 10 mg kg 1 rifampicin intraperitoneally (i.p.) at 1 day post infection (dpi)

The activation of Nrf2 has been shown to reduce bisphenol A-induced lipid accumulation in the liver of mice, and its efficacy in inhibiting lipid accumulation is considered to be related to the enhanced expression of genes coding for antioxidants and detoxification enzymes (Ludtmann et al., 2014

Among them, compounds 112-114 showed low nanomole inhibitory activities against IKK and good pharmacokinetics [191]