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[] METTL4 is localized to mitochondria, preferentially methylates promoter regions of mtDNA, and disrupts assembly of the transcription initiation complex, leading to transcriptional stalling, impaired electron transport chain (ETC) genes expression, mitochondrial respiratory deficiency, and ultimately HF

(128) have reported that reserpine-induced decreases in DA resulted in a significant increase in the expression of AT 1 and AT 2 receptors