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The result could be: Higher baseline mitochondrial capacity (SLU-PP-332) Stronger demand to use that capacity and burn fat/glucose (ATX-304) Potentially additive or synergistic effects on energy expenditure and adiposity endpoints Because AMPK and ERR/PGC-1 are known to cross-talk in muscle and liver, combining a pan-AMPK activator and an ERR agonist is a logical way to probe multi-node metabolic reprogramming in preclinical work