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It is highly expressed on plasmablasts, plasma cells, and memory B cells, with particularly elevated levels observed on LLPCs in the bone marrow (94) ( Figure 2 )

Future directions The results presented in this study suggest that targeting fundamental biological mechanisms holds promise for the future development of ASD therapies

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GPX4 overexpression in ALS reduces the cytotoxicity of SOD1, delays ALS onset, improves motor neuron function, and prolongs survival (Chen et al., Multiple studies have demonstrated the treatment of ALS by inhibiting iron death in motor neurons