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In the present study, we further demonstrate that BPC 157 can promote the phosphorylation of Src and Cav-1, providing a strong explanation for the activation and endocytosis of VEGFR2 by BPC 157 as reported in our previous study

Additionally, the peptide might have reactivated the apoptosis pathway, a type of programmed cell death crucial for removing faulty cells, as evidenced by activity in caspases 3 and 7, which are enzymes that play key roles in apoptosis

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Pharmacologic approaches to glycemic treatment: Standards of care in diabetes2026