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Amniotic MSCs-derived EXOs promote trophoblast proliferation by blocking the EZH2/mTOR signaling pathway, which in turn enhances autophagy in trophoblasts (Chu et al., 2020)

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Hence, when iron hemostasis is interrupted, excessive iron may injure proteins, lipids and DNA within mitochondria and reduce ATP production, leading to energy stress and ferroptosis (Fig