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however, liver toxicity, expressed as elevated liver enzymes, was much lower in patients treated with NAC

Hypoxia and inflammation activate p53 via phosphorylation

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In summary, the present study revealed that early accumulation of iA, coinciding with a neuron-derived inflammatory response (as previously shown 36 ) also coincides with altered expression of oxidative stress-related genes including key DNA repair and antioxidant genes, where not all gene level alterations were reflected at the protein level