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Consequently, we aim to prevent the progression of atrial fibrillation by either suppressing the production of IsoLGs or enhancing their clearance process to inhibit ferroptosis in the myocardium, improving the prognosis of patients with AF

Knockdown of GPx1 in macrophages from healthy volunteers resulted in increased H 2 O 2 -induced cell death reminiscent of that observed with macrophages from CD

The reduction of Cu 2 in the presence of neocuproine by a reducing agent produces a Cu complex, which exhibits a maximum absorption peak at 450 nm (Tutem et al

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