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The mechanistic rationale builds on earlier work by Kraus and colleagues in Nature (2014), in which adipocyte-specific NNMT knockdown protected mice from diet-induced obesity, establishing NNMT as a metabolic-disease target candidate [2]

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Growing evidence indicates that bisphenol-A can modify the neurogenic process, affecting the proliferation/differentiation balance of NSPCs and neuronal maturation

Kyoto Encyclopedia of Genes and Genomes (KEGG) analysis of the DEGs revealed that many were involved in inflammation (Fig