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These findings suggest that neuronal cells in AD disease upregulate ferritin and downregulate FPN expression, increasing iron intake and reducing iron excretion

No statistically significant increases in chromosomal aberrations were observed in CHO cells at any tested concentration

These developments are consistent with the evolving understanding of the gastrointestinal tract as a key endocrine organ that regulates appetite, glucose homeostasis, energy expenditure, and gastrointestinal motility through a complex network of hormonal signals [20]

For RCTs, this included assessment of: (1) risk of bias (via Cochrane RoB 2 tool), (2) inconsistency of results across studies, (3) indirectness of evidence, (4) imprecision due to sample size or confidence intervals, and (5) potential publication bias