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Using the second harmonic generation properties of fibrilar collagen and multi-photon microscopy, we confirmed that fibroblasts from former smokers with COPD were unable to efficiently remodel collagen into fibrils (Figure 6d)

Furthermore, the progression of AH to ACLF is influenced by epigenetic modifications, and the development of hepatocellular failure in AH patients is characterized by a significant reduction in the hepatocyte nuclear factor 4 alpha (HNF4)-dependent gene expression