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Abnormal NLRP3 inflammasome activation and excessive IL-1 secretion is the major reason of CAPS, and has been demonstrated in three clinical subtypes: neonatal-onset multisystem inflammatory disease (NOMID), Muckle-Wells syndrome (MWS), and familial cold autoinflammatory syndrome (FCAS)

In one study, the cell-to-cell interaction between HUVEC and QG90 cell, a tumor cell line derived from human small cell carcinoma of the lung expressing sialyl-LewisX antigen was studied, and it was found that IL-1 was able to induce the attachment of cancer cells to HUVEC

Oxidative stress happens when free radicals start to outpace your antioxidant defenses

However, mechanistic findings from preclinical studiessuch as the neuroprotective effects of TA in alleviating Cd-induced oxidative damage, the ability of Ca-EDTA to enhance thrombolysis by chelating Zn, and the role of RNAi in regulating Cu homeostasisprovide sufficient basis for subsequent clinical exploration