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demonstrated that oxidative stress and H 2 O 2 treatment led to a marked increase in senescence-associated -galactosidase activity but only to minimal apoptotic cell death in CSCs, suggesting that ROS triggered the induction of senescence and exhibited therapeutic potential in the eradication of drug-resistant CSCs [8]

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We show that the stereochemical identity of lipids influences LNP structure and has a major impact on delivery efficacy, establishing a direct link between stereochemistry, LNP architecture, and biological performance

Lipopolysaccharide (LPS)-induced inflammatory responses, as the most representative septic pathway, initiate a premise for immune cell mobilization and are a potent driver of this metabolic reprogramming