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employed Western blot analysis to demonstrate that HIF-1 expression in myocardial tissues from the I/R group peaked at 2 h post-reperfusion, with concurrent upregulation of TfR and Tf levels mirroring the HIF-1 elevation [85]

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Both in vitro and in vivo experiments have demonstrated that inhibition of CI and disruption of the respiratory super complex in the Her2-positive background, leads to increased production of ROS material and cell death