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It can be hypothesised that the incidence of DNA damage due to mitochondrial dysfunction in oocytes can be reduced by supplementation with antioxidants, for example

However, acetylating the N-terminal end of Cys, thus creating the compound NAC, increases the stability of the molecule and allows for more efficient delivery of reduced sulfhydryl moieties [28]

Relative DNA methylation, chromatin accessibility, and histone modifications at the Casp1 promoter site comparing Nave, Resolved and Sensitized USCs are shown as a heatmap by displaying the normalized signal using read coverage and fraction of reads under consensus peaks

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