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By modulating M1 macrophage activity and reducing inflammation, the anti-M1 agent indirectly limits the profibrotic activity of M2 macrophages, thereby reducing the excessive accumulation of ECM and preventing fibrosis 25,53

3i), which suggests that increased 2-AB reflected a compensation in GSH levels for consumption by ROS

Cell Biochem BiophysMechanism of the KIAA1429/KLF1/PD-L1 Axis in Regulating Immune Escape in Non-small Cell Lung Cancer

A higher production of reactive oxygen metabolites, such as the superoxide anion, affects catalase activity 45 , and an increase in production of hydroxyl ions reduces catalase activity 46