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Indeed, it can be triggered by the perturbation of a variety of systems known to be implicated in schizophrenia, which include the redox, neuroimmune, glutamatergic, and dopaminergic systems 1,2

Consequently, its logical therapeutical applications seems to be limited to restore physiological circulating levels in order to recover the clinical consequences of IGF-I deficiency, conditions where, despite continuous discrepancies, IGF-I treatment has never been related to oncogenesis

Precise regulation of differential distribution or expression of cytokines or signaling in intracellular and even subcellular structures may play a critical role in regulating FSP1-mediated programmed cell death

First, antibiotics alter the host immune system indirectly by affecting the host microbiota composition (108)